Category: Blog

PTPN5 promotes follicle-stimulating hormone secretion through regulating intracellular calcium homeostasis

PTPN5 promotes follicle-stimulating hormone secretion through regulating intracellular calcium homeostasisPTPN5 promotes follicle-stimulating hormone secretion through regulating intracellular calcium homeostasis

Protein tyrosine phosphatase non-receptor kind 5 (PTPN5), additionally known as striatal-enriched protein tyrosine phosphatase (STEP), is very expressed in neurons of the basal ganglia, hippocampus, cortex, and associated constructions, additionally

The antibiotic robenidine exhibits guanabenz-like cytoprotective properties by a mechanism independent of protein phosphatase PP1:PPP1R15A.

The antibiotic robenidine exhibits guanabenz-like cytoprotective properties by a mechanism independent of protein phosphatase PP1:PPP1R15A.The antibiotic robenidine exhibits guanabenz-like cytoprotective properties by a mechanism independent of protein phosphatase PP1:PPP1R15A.

The aminoguanidine compound robenidine is extensively used as an antibiotic for the management of coccidiosis, a protozoal an infection in poultry and rabbits. Apparently, robenidine is structurally much like guanabenz

Identification of Androgen Receptor Metabolic Correlome Reveals the Repression of Ceramide Kinase by Androgens

Identification of Androgen Receptor Metabolic Correlome Reveals the Repression of Ceramide Kinase by AndrogensIdentification of Androgen Receptor Metabolic Correlome Reveals the Repression of Ceramide Kinase by Androgens

Prostate most cancers (PCa) is likely one of the most prevalent cancers in males. Androgen receptor signaling performs a serious function on this illness, and androgen deprivation remedy is a

Generation of an induced pluripotent stem cell line from a Bartter syndrome patient with the homozygote mutation p.A244D (c.731C>A) in SLC12A1 gene

Generation of an induced pluripotent stem cell line from a Bartter syndrome patient with the homozygote mutation p.A244D (c.731C>A) in SLC12A1 geneGeneration of an induced pluripotent stem cell line from a Bartter syndrome patient with the homozygote mutation p.A244D (c.731C>A) in SLC12A1 gene

Bartter Syndrome (BS) is a gaggle of uncommon inherited autosome-recessive illness, which might be attributable to the gene mutations of sodium-potassium-chloride cotransporter gene (SLC12A1). Right here, the urine cells (UCs)

P23 Acts as Functional RBP in the Macrophage Inflammation Response
S100A4/non-muscle myosin II signaling regulates epithelial-mesenchymal transition and stemness in uterine carcinosarcoma.

S100A4/non-muscle myosin II signaling regulates epithelial-mesenchymal transition and stemness in uterine carcinosarcoma.S100A4/non-muscle myosin II signaling regulates epithelial-mesenchymal transition and stemness in uterine carcinosarcoma.

Uterine carcinosarcoma (UCS) represents a real instance of most cancers related to epithelial-mesenchymal transition (EMT), which displays most cancers stem cell (CSC)-like traits. Though S100A4 is an inducer of EMT, little is